HOW ALUMINUM DAMAGES YOUR BRAIN

For years, I’ve warned that aluminum is a serious neurotoxic hazard involved in rising rates of autism and Alzheimer’s disease (AD). I’ve also warned that vaccines are a significant source of such exposure, and may be one of the worst, since by injecting it, the aluminum bypasses your body’s natural filtering and detoxification systems.

My comments above were one of the reasons the self-appointed global arbiter of fake news, NewsGuard, refused to give us “green” status as a site that follows “basic standards of accuracy and accountability.” In other words, our reporting of aluminum hazards was deemed “fake news.”

Not only were my earlier reports based on published science, but now we have yet another study, published in the Journal of Alzheimer’s Disease, strongly linking aluminum exposure to AD. As reported by SciTech Daily:

“Researchers found significant amounts of aluminum content in brain tissue from donors with familial AD. The study also found a high degree of co-location with the amyloid-beta protein, which leads to early onset of the disease.

 

‘This is the second study confirming significantly high brain accumulation in familial Alzheimer’s disease, but it is the first to demonstrate an unequivocal association between the location of aluminum and amyloid-beta in the disease.

 

It shows that aluminum and amyloid-beta are intimately woven in the neuropathology,’ explained lead investigator Christopher Exley, PhD, Birchall Centre, Lennard-Jones Laboratories, Keele University, Staffordshire, UK.”

The Association Between Aluminum and Amyloid-Beta

To gain a better understanding of the link between aluminum exposure and beta-amyloid generation, the researchers examined the brain tissue of donors diagnosed with familial Alzheimer’s disease who also had a specific gene mutation known to increase levels of amyloid-beta, leading to early onset and more aggressive disease.

Aluminum levels were compared to controls with no neurological disease diagnosis. They found striking differences between these two groups. Donors with the genetic mutation had universally high aluminum content.

While all samples had some level of aluminum, 42% of the samples from those with familial Alzheimer’s had “pathologically significant” aluminum levels, and the aluminum was primarily co-located with amyloid beta plaques. As reported by SciTech Daily:

“The results strongly suggest that genetic predispositions known to increase amyloid-beta in brain tissue also predispose individuals to accumulate and retain aluminum in brain tissue …

 

‘One could envisage increased amyloid-beta in brain tissue as a response to high levels of aluminum content, or that aluminum fosters the accumulation of amyloid-beta,’ said Dr. Exley.

 

‘Either way, the new research confirms my resolve that within the normal lifespan of humans, there would not be any AD if there were no aluminum in the brain tissue. No aluminum, no AD.’”

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