The spike protein binds to human ACE2 receptors and also causes cellular damage. This has an effect on what’s known as the renin-angiotensin system. This system plays a role in many important biological functions. I think they are hiding the long term effects of COVID as well as the long term effects of the vaccine.
I’m going to make an educated guess based on all the data cited below about delayed symptoms of COVID as well as the vaccine which we could see in the future.
- Blindness
- Diabetes
- Erectile dysfunction
- Mental health disorders caused directly by the spike protein
- Menstrual irregularities
- Malformed and damaged ovum
- Lack of strength, lack of body building muscle in response to exercise
- Premature aging
- Kidney fibrosis
- Issues with T-cell differentiation (AIDS symptoms)
- Central nervous system disorders caused by inflammation
This is not the whole list, just a start!
Below are the titles and links to scientific articles which back up my assumptions. They are all credible sources of information.
No company has publicly published their biodistribution studies, however we have the below.
This is the EMA (European Medical Association) review of the company data they had access to for “Comirnaty” (Covid-19 mRNA vaccine) which included animal biodistribution studies
(The biodistribution was also studied in rats using radiolabeled LNP and luciferase modRNA (study 185350). The radiolabeling data, measuring distribution to blood, plasma and selected tissues, of IM injection of a single dose of 50 μg mRNA over a 48-hour period is considered more sensitive than the bioluminescence method and indicate a broader biodistribution pattern than was observed with bioluminescence. Over 48 hours, distribution from the injection site to most tissues occurred, with the majority of tissues exhibiting low levels of radioactivity.)
So, basically, they made the mRNA radioactive so they could detect it and checked where they could find it 48 hours after vaccinating rats. They gave the rats an intramuscular injection which is the same type of injection they are giving people now. It ended up in almost all of their tissues.
This proves that, contrary to popular belief and what the media is spouting, the mRNA does not stay in the muscle and travels throughout the whole body!
This also proves the mRNA will create spike proteins anywhere ACE2 receptors exist in the entire body, which is how it could potentially effect all the systems mentioned below.
The problem with this is that once a spike protein binds to an ACE2 expressing cell (angiotensin converting enzyme II receptor) it prevents the angiotensin circulating from being picked up by the receptor. There are trillions of mRNA fragments in a single dose of vaccine. This can cause a disruption to your renin-angiotensin system.
SARS-CoV-2 vaccines: Lights and shadows
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8084611/
(The resulting pathological features may resemble those of active coronavirus disease. The free-floating Spike proteins synthetized by cells targeted by vaccine and destroyed by the immune response circulate in the blood and systematically interact with angiotensin converting enzyme 2 (ACE2) receptors expressed by a variety of cells including platelets, thereby promoting ACE2 internalization and degradation.)
The above also states that when a vaccinated cell dies or is destroyed by the immune system, the debris may release a large amount of Spike proteins and protein fragments (free-floating Spike proteins). Which can then bind to ACE2 expressing cells.
Estradiol modulation of the renin-angiotensin system and the regulation of fear extinction
https://pubmed.ncbi.nlm.nih.gov/30696810/
(Giving rats drugs that altered the renin-angiotensin system helped de-condition them from previously taught triggers, essentially the equivalent of helping them get over PTSD)
The renin-angiotensin system: a possible new target for depression
https://pubmed.ncbi.nlm.nih.gov/28760142/
(Drugs blocking the angiotensin system have efficacy in several animal models of depression.)
The effect of combined treatment with SSRIs and renin-angiotensin system (RAS) drugs: A propensity score matched cohort study
https://pubmed.ncbi.nlm.nih.gov/32001138/
(Drugs acting on the renin-angiotensin system (RAS) may have beneficial effects on mental health.)
Linking Meditation with Improved Mental Health
I included this article because it ties in the importance of the RAS system and mental wellbeing.
https://www.womenmeanbusiness.com/2020/02/linking-meditation-with-improved-mental-health/
(Although the scope of research is currently limited, it seems that meditation may also influence the RAA system, corresponding with improved well-being and changes in hormonal stress.”)
High blood pressure drugs impact depression, bipolar disorder
https://www.medicalnewstoday.com/articles/313380
(In contrast, patients prescribed angiotensin antagonists had the lowest risk for hospitalization with mood disorders, compared with patients taking other blood pressure drugs)
Exploring the Evidence Implicating the Renin-Angiotensin System (RAS) in the Physiopathology of Mood Disorders
https://www.eurekaselect.com/177717/article
(“Low levels of RAS components (renin activity or aldosterone) and mood symptoms improvement with ACE inhibitors or AT1 blockers were also observed in mood disorders. Overall, this review reiterates the strong and under-researched connection between RAS and mood disorders.”)
Associations of trauma exposure and post-traumatic stress disorder with the activity of the renin–angiotensin–aldosterone-system in the general population
(Finding elevated renin levels in PTSD independent from trauma load supports the concept of PTSD as a disorder with specific neuroendocrine characteristics.)
Association between anxiety and hypertension: a systematic review and meta-analysis of epidemiological studies
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4411016/
(Second, anxiety has a close relationship with the renin angiotensin system and increases the level of angiotensin II)
System That Regulates Blood Pressure May Also Affect Aging
https://www.sciencedaily.com/releases/2004/11/041122095829.htm
(At the end of the program, however, participants with the combination associated with highest ACE production showed a 75 percent improvement in knee strength – compared to a 23 percent improvement in participants who had the combination associated with lowest ACE production)
An Interview with Björn Folkow, M.D., Ph.D.
https://www.stress.org/stress-hypertension-and-the-metabolic-syndrome
(These and other drugs like ACE inhibitors that inhibit the renin-angiotensin system have also been shown to significantly reduce the incidence of Type 2 diabetes in patients with hypertension and congestive failure.)
Role of ACE inhibitors in patients with diabetes mellitus
https://pubmed.ncbi.nlm.nih.gov/11708761/
(Finally, there are a growing number of arguments favouring the use of ACE inhibitors very early in patients with diabetes mellitus.)
Diminazene protects corpus cavernosum against hypercholesterolemia-induced injury
https://pubmed.ncbi.nlm.nih.gov/25411084/
(ACE2 is expressed in erectile tissue and its reduction is associated with hypercholesterolemia-induced ED)
Research sheds light on the role of ACE2 in ovulation
https://www.bionews.org.uk/page_158735
(This research suggests the potential involvement of ACE2 as a critical enzyme for the LH surge-induced cyclic events of ovulation.)
The retinal renin-angiotensin system: roles of angiotensin II and aldosterone
https://pubmed.ncbi.nlm.nih.gov/22537944/
(The therapeutic utility of inhibiting key components of the renin-angiotensin system is complex, but may hold promise for the prevention and improvement of vision threatening diseases.)
The renin-angiotensin system and the retinal neurovascular unit: A role in vascular regulation and disease
https://pubmed.ncbi.nlm.nih.gov/31408629/
(The retina is known to have a local renin-angiotensin system (RAS) and dysfunction in the RAS is often associated with diseases of the retinal vasculature that cause irreversible vision loss.)
Role of the renin-angiotensin system in autoimmune inflammation of the central nervous system
https://pubmed.ncbi.nlm.nih.gov/19706425/
(Treatment with the renin inhibitor aliskiren, the angiotensin II converting-enzyme inhibitor enalapril, as well as preventive or therapeutic application of the AT1R antagonist losartan, resulted in a significantly ameliorated course of MOG-EAE)
Role of renin-angiotensin system in inflammation, immunity and aging
https://pubmed.ncbi.nlm.nih.gov/22283774/
(Classic RAS blockers have been proposed as anti-inflammatory and immunomodulatory agents and some studies suggest a new potential application of RAS blockers in autoimmune diseases.)
Angiotensin II Regulates Th1 T Cell Differentiation Through Angiotensin II Type 1 Receptor-PKA-Mediated Activation of Proteasome
https://pubmed.ncbi.nlm.nih.gov/29462804/
(This study for the first time demonstrates that Ang II activates AT1R-PKA-proteasome pathway, which promotes degradation of IκBα and MKP-1 and activation of STAT1 and NF-κB thereby leading to Th1 differentiation. Thus, inhibition of proteasome activation might be a potential therapeutic target for Th1-mediated diseases.)
The above issue of a lack of T-cell differentiation is also a result of AIDS as seen in this next link.
T cell selection and differentiation in AIDS disease: the model of HIV-discordant monozygotic twins
https://pubmed.ncbi.nlm.nih.gov/18088551/
(In our HIV-discordant monozygotic twins, a significant reduction of naive T cells and a parallel accumulation of effector/memory T cells was induced by HIV infection)
The renin-angiotensin system: an old, newly discovered player in immunoregulation
https://pubmed.ncbi.nlm.nih.gov/19539879/
(Of special clinical interest is the ability of the RAS to activate the transforming growth factor beta(1) and the Smad pathways leading to fibrinogenesis)
